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Difference between revisions of "Krako Jakovljevic 2021 BEC PD"

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|title=Krako Jakovljevic N, Ebanks B, Katyal G, Chakrabarti L, Markovic I, Moisoi N (2021) Mitochondrial homeostasis in cellular models of Parkinson’s Disease. Bioenerg Commun 2021.2. [[doi:10.26124/bec:2021-0002]]
|title=Krako Jakovljevic N, Ebanks B, Katyal G, Chakrabarti L, Markovic I, Moisoi N (2021) Mitochondrial homeostasis in cellular models of Parkinson’s Disease. Bioenerg Commun 2021.2. [[doi:10.26124/bec:2021-0002]]


|info=[[File:OpenAccess-downloadPDF.png|240px||link=https://www.bioenergetics-communications.org/index.php/bec/article/view/krako_jakovljevic_2021_pd |Open Access pdf]]  ''Published online:'' 2021-Oct-06<br /><br />
|info=[[File:OpenAccess-downloadPDF.png|240px||link=https://www.bioenergetics-communications.org/index.php/bec/article/view/krako_jakovljevic_2021_pd/27 |Open Access pdf]]  ''Published online:'' 2021-Oct-06<br /><br />


|authors=Krako Jakovljevic Nina, Ebanks Brad, Katyal Gunjan, Chakrabarti Lisa, Markovic Ivanka, Moisoi Nicoleta
|authors=Krako Jakovljevic Nina, Ebanks Brad, Katyal Gunjan, Chakrabarti Lisa, Markovic Ivanka, Moisoi Nicoleta

Revision as of 10:06, 3 November 2021


Bioenergetics Communications        
Gnaiger 2020 BEC MitoPathways
       
Gnaiger Erich et al ― MitoEAGLE Task Group (2020) Mitochondrial physiology. Bioenerg Commun 2020.1.
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Bioenergetics Communications
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Publications in the MiPMap
Krako Jakovljevic N, Ebanks B, Katyal G, Chakrabarti L, Markovic I, Moisoi N (2021) Mitochondrial homeostasis in cellular models of Parkinson’s Disease. Bioenerg Commun 2021.2. doi:10.26124/bec:2021-0002

» Open Access pdf Published online: 2021-Oct-06

Krako Jakovljevic Nina, Ebanks Brad, Katyal Gunjan, Chakrabarti Lisa, Markovic Ivanka, Moisoi Nicoleta (2021-10-06) Bioenerg Commun

Abstract: Mitochondrial function is known to be an important factor in maintaining cellular homeostasis and its dysregulation has become a hallmark for multiple disease conditions. This review aims to sythesise the extent of this knowledge by analysing changes of mitochondrial physiology parameters in Parkinson’s disease (PD) and to evaluate the contribution of cellular models of PD in the field. The analysis provided here constitutes a platform for further elucidation of mitochondrial function parameters relative to factors that may potentiate disease progression. Keywords: ATP, mitochondrial homeostasis, mitochondrial respiration, Parkinson’s disease, cellular models of Parkinson’s, qualitative analysis Bioblast editor: Gnaiger E, Cardoso LHD O2k-Network Lab: RS Belgrade Lalic NM, UK Nottingham Chakrabarti L, UK Leicester Moisoi N


ORCID: ORCID.png Krako Jakovljevic Nina, ORCID.png Ebanks Brad, ORCID.png Katyal Gunjan, ORCID.png Chakrabarti Lisa, ORCID.png, Markovic Ivanka ORCID.png Moisoi Nicoleta



Preprint

Krako Jakovljevic 2021 MitoFit PD


Labels: Pathology: Parkinson's 

Organism: Human, Mouse, Drosophila